Journal of Diagnostics Concepts & Practice ›› 2019, Vol. 18 ›› Issue (04): 383-386.doi: 10.16150/j.1671-2870.2019.04.002
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Received:2019-08-05
Online:2019-08-25
Published:2019-08-25
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| [1] | 郑荣寿, 孙可欣, 张思维, 等. 2015年中国恶性肿瘤流行情况分析[J]. 中华肿瘤杂志, 2019, 41(1):19-28. |
| [2] |
Aleksandrova K, Pischon T, Jenab M, et al. Combined impact of healthy lifestyle factors on colorectal cancer: a large European cohort study[J]. BMC Med, 2014, 12:168.
doi: 10.1186/s12916-014-0168-4 pmid: 25319089 |
| [3] |
Knudsen MD, de Lange T, Botteri E, et al. Favorable lifestyle before diagnosis associated with lower risk of screen-detected advanced colorectal neoplasia[J]. World J Gastroenterol, 2016, 22(27):6276-6286.
doi: 10.3748/wjg.v22.i27.6276 URL |
| [4] |
Chan DS, Lau R, Aune D, et al. Red and processed meat and colorectal cancer incidence: meta-analysis of prospective studies[J]. PLoS One, 2011, 6(6):e20456.
doi: 10.1371/journal.pone.0020456 URL |
| [5] |
Xu X, Yu E, Gao X, et al. Red and processed meat intake and risk of colorectal adenomas: a meta-analysis of observational studies[J]. Int J Cancer, 2013, 132(2):437-448.
doi: 10.1002/ijc.27625 URL |
| [6] | Cross AJ, Pollock JR, Bingham SA. Haem, not protein or inorganic iron, is responsible for endogenous intestinal N-nitrosation arising from red meat[J]. Cancer Res, 2003, 63(10):2358-2360. |
| [7] |
Santarelli RL, Pierre F, Corpet DE. Processed meat and colorectal cancer: a review of epidemiologic and experimental evidence[J]. Nutr Cancer, 2008, 60(2):131-144.
doi: 10.1080/01635580701684872 pmid: 18444144 |
| [8] | Zeng H, Umar S, Rust B, et al. Secondary Bile Acids and Short Chain Fatty Acids in the Colon: A Focus on Colonic Microbiome, Cell Proliferation, Inflammation, and Cancer[J]. Int J Mol Sci, 2019, 20(5): pii: E1214. |
| [9] |
Ajouz H, Mukherji D, Shamseddine A. Secondary bile acids: an underrecognized cause of colon cancer[J]. World J Surg Oncol, 2014, 12:164.
doi: 10.1186/1477-7819-12-164 pmid: 24884764 |
| [10] |
Bernstein H, Bernstein C, Payne CM, et al. Bile acids as carcinogens in human gastrointestinal cancers[J]. Mutat Res, 2005, 589(1):47-65.
doi: 10.1016/j.mrrev.2004.08.001 URL |
| [11] |
Aune D, Chan DS, Lau R, et al. Dietary fibre, whole grains, and risk of colorectal cancer: systematic review and dose-response meta-analysis of prospective studies[J]. BMJ, 2011, 343:d6617.
doi: 10.1136/bmj.d6617 URL |
| [12] |
Lattimer JM, Haub MD. Effects of dietary fiber and its components on metabolic health[J]. Nutrients, 2010, 2(12):1266-1289.
doi: 10.3390/nu2121266 pmid: 22254008 |
| [13] |
Donohoe DR, Collins LB, Wali A, et al. The Warburg effect dictates the mechanism of butyrate-mediated histone acetylation and cell proliferation[J]. Mol Cell, 2012, 48(4):612-626.
doi: 10.1016/j.molcel.2012.08.033 pmid: 23063526 |
| [14] | Davie JR. Inhibition of histone deacetylase activity by butyrate[J]. J Nutr, 2003, 133(7 Suppl):2485S-2493S. |
| [15] | Cao Y, Gao X, Zhang W, et al. Dietary fiber enhances TGF-β signaling and growth inhibition in the gut[J]. Am J Physiol Gastrointest Liver Physiol, 2011, 301(1):G156-G164. |
| [16] |
Bordonaro M, Lazarova DL. CREB-binding protein, p300, butyrate, and Wnt signaling in colorectal cancer[J]. World J Gastroenterol, 2015, 21(27):8238-8248.
doi: 10.3748/wjg.v21.i27.8238 URL |
| [17] |
Lazarova DL, Chiaro C, Wong T, et al. CBP Activity Mediates Effects of the Histone Deacetylase Inhibitor Butyrate on WNT Activity and Apoptosis in Colon Cancer Cells[J]. J Cancer, 2013, 4(6):481-490.
doi: 10.7150/jca.6583 pmid: 23901348 |
| [18] |
Fedirko V, Tramacere I, Bagnardi V, et al. Alcohol drinking and colorectal cancer risk: an overall and dose-response meta-analysis of published studies[J]. Ann Oncol, 2011, 22(9):1958-1972.
doi: S0923-7534(19)38342-5 pmid: 21307158 |
| [19] |
Matsuda T, Yabushita H, Kanaly RA, et al. Increased DNA damage in ALDH2-deficient alcoholics[J]. Chem Res Toxicol, 2006, 19(10):1374-1378.
doi: 10.1021/tx060113h URL |
| [20] | Bishehsari F, Magno E, Swanson G, et al. Alcohol and Gut-Derived Inflammation[J]. Alcohol Res, 2017, 38(2):163-171. |
| [21] |
Shukla PK, Chaudhry KK, Mir H, et al. Chronic ethanol feeding promotes azoxymethane and dextran sulfate so-dium-induced colonic tumorigenesis potentially by enhancing mucosal inflammation[J]. BMC Cancer, 2016, 16:189.
doi: 10.1186/s12885-016-2180-x URL |
| [22] | Zakhari S. Alcohol metabolism and epigenetics changes[J]. Alcohol Res, 2013, 35(1):6-16. |
| [23] | Kim YI. Role of folate in colon cancer development and progression[J]. J Nutr, 2003, 133(11 Suppl 1):3731S-3739S. |
| [24] |
Kelemen LE. The role of folate receptor alpha in cancer development, progression and treatment: cause, consequence or innocent bystander?[J]. Int J Cancer, 2006, 119(2):243-250.
pmid: 16453285 |
| [25] |
Carroll C, Cooper K, Papaioannou D, et al. Meta-analysis: folic acid in the chemoprevention of colorectal adenomas and colorectal cancer[J]. Aliment Pharmacol Ther, 2010, 31(7):708-718.
doi: 10.1111/j.1365-2036.2010.04238.x URL |
| [26] |
Moazzen S, Dolatkhah R, Tabrizi JS, et al. Folic acid intake and folate status and colorectal cancer risk: A systematic review and meta-analysis[J]. Clin Nutr, 2018, 37(6 Pt A):1926-1934.
doi: S0261-5614(17)31383-3 pmid: 29132834 |
| [27] |
Yin L, Grandi N, Raum E, et al. Meta-analysis: longitudinal studies of serum vitamin D and colorectal cancer risk[J]. Aliment Pharmacol Ther, 2009, 30(2):113-125.
doi: 10.1111/j.1365-2036.2009.04022.x URL |
| [28] |
Larriba MJ, Ordóñez-Morán P, Chicote I, et al. Vitamin D receptor deficiency enhances Wnt/β-catenin signaling and tumor burden in colon cancer[J]. PLoS One, 2011, 6(8):e23524.
doi: 10.1371/journal.pone.0023524 URL |
| [29] |
Ferrer-Mayorga G, Larriba MJ, Crespo P, et al. Mechanisms of action of vitamin D in colon cancer[J]. J Steroid Biochem Mol Biol. 2019 Jan; 185:1-6.
doi: 10.1016/j.jsbmb.2018.07.002 URL |
| [30] |
Hopkins MH, Owen J, Ahearn T, et al. Effects of supplemental vitamin D and calcium on biomarkers of inflammation in colorectal adenoma patients: a randomized, controlled clinical trial[J]. Cancer Prev Res (Phila), 2011, 4(10):1645-1654.
doi: 10.1158/1940-6207.CAPR-11-0105 URL |
| [31] |
Protiva P, Pendyala S, Nelson C, et al. Calcium and 1,25-dihydroxyvitamin D3 modulate genes of immune and inflammatory pathways in the human colon: a human crossover trial[J]. Am J Clin Nutr, 2016, 103(5):1224-1231.
doi: 10.3945/ajcn.114.105304 pmid: 27009752 |
| [32] |
Cruzalegui FH, Bading H. Calcium-regulated protein kinase cascades and their transcription factor targets[J]. Cell Mol Life Sci, 2000, 57(3):402-410.
pmid: 10823241 |
| [33] |
Bonithon-Kopp C, Kronborg O, Giacosa A, et al. Calcium and fibre supplementation in prevention of colorectal adenoma recurrence: a randomised intervention trial. European Cancer Prevention Organisation Study Group[J]. Lancet. 2000 Oct 14; 356(9238):1300-1306.
pmid: 11073017 |
| [34] |
Baron JA, Beach M, Mandel JS, et al. Calcium supplements for the prevention of colorectal adenomas. Calcium Polyp Prevention Study Group[J]. N Engl J Med, 1999, 340(2):101-107.
doi: 10.1056/NEJM199901143400204 URL |
| [35] |
Combs GF Jr, Gray WP. Chemopreventive agents: selenium[J]. Pharmacol Ther, 1998, 79(3):179-192.
doi: 10.1016/S0163-7258(98)00014-X URL |
| [36] |
Bonelli L, Puntoni M, Gatteschi B, et al. Antioxidant supplement and long-term reduction of recurrent adenomas of the large bowel. A double-blind randomized trial[J]. J Gastroenterol, 2013, 48(6):698-705.
doi: 10.1007/s00535-012-0691-z URL |
| [37] |
Reid ME, Duffield-Lillico AJ, Sunga A, et al. Selenium supplementation and colorectal adenomas: an analysis of the nutritional prevention of cancer trial[J]. Int J Cancer, 2006, 118(7):1777-1781.
doi: 10.1002/ijc.21529 URL |
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